Открыть главное меню
Главная
Случайная
Войти
Настройки
О hpluswiki
Отказ от ответственности
hpluswiki
Найти
Редактирование:
Aging genes 60-69
(раздел)
Внимание:
Вы не вошли в систему. Ваш IP-адрес будет общедоступен, если вы запишете какие-либо изменения. Если вы
войдёте
или
создадите учётную запись
, её имя будет использоваться вместо IP-адреса, наряду с другими преимуществами.
Анти-спам проверка.
Не
заполняйте это!
==LMNA== * {{medline-title |title=Metformin alters peripheral blood mononuclear cells (PBMC) senescence biomarkers gene expression in type 2 diabetic patients. |date=01.2021 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/33187870 |full-text-url=https://sci-hub.do/10.1016/j.jdiacomp.2020.107758 }} * {{medline-title |title=Protein structural and mechanistic basis of progeroid laminopathies. |date=16.08.2020 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/32799420 |full-text-url=https://sci-hub.do/10.1111/febs.15526 }} * {{medline-title |title=Towards delineating the chain of events that cause premature senescence in the accelerated aging syndrome Hutchinson-Gilford progeria (HGPS). |date=30.06.2020 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/32539085 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7329345 }} * {{medline-title |title=Progerin Expression Induces Inflammation, Oxidative Stress and Senescence in Human Coronary Endothelial Cells. |date=12.05.2020 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/32408587 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7290406 }} * {{medline-title |title=The JAK1/2 inhibitor ruxolitinib delays premature aging phenotypes. |date=04.2020 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/32196928 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7189991 }} * {{medline-title |title=Pharmacotherapy to gene editing: potential therapeutic approaches for Hutchinson-Gilford progeria syndrome. |date=04.2020 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/32048129 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7205988 }} * {{medline-title |title=Long term breeding of the Lmna G609G progeric mouse: Characterization of homozygous and heterozygous models. |date=02.2020 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/31794853 |full-text-url=https://sci-hub.do/10.1016/j.exger.2019.110784 }} * {{medline-title |title=Epigenetic signatures of Werner syndrome occur early in life and are distinct from normal epigenetic aging processes. |date=10.2019 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/31259468 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6718529 }} * {{medline-title |title=Transient introduction of human telomerase mRNA improves hallmarks of progeria cells. |date=08.2019 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/31152494 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6612639 }} * {{medline-title |title=A humanized yeast system to analyze cleavage of prelamin A by [[ZMPSTE24]]. |date=15.03.2019 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/30625386 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6401292 }} * {{medline-title |title=Impaired immune surveillance accelerates accumulation of senescent cells and aging. |date=21.12.2018 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/30575733 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6303397 }} * {{medline-title |title=Endothelial progerin expression causes cardiovascular pathology through an impaired mechanoresponse. |date=01.02.2019 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/30422822 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6355303 }} * {{medline-title |title=Targeting the phospholipase A2 receptor ameliorates premature aging phenotypes. |date=12.2018 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/30216637 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6260922 }} * {{medline-title |title=Altered modulation of lamin A/C-[[HDAC2]] interaction and p21 expression during oxidative stress response in HGPS. |date=10.2018 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/30109767 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6156291 }} * {{medline-title |title=Cellular stress and AMPK activation as a common mechanism of action linking the effects of metformin and diverse compounds that alleviate accelerated aging defects in Hutchinson-Gilford progeria syndrome. |date=09.2018 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/30037605 |full-text-url=https://sci-hub.do/10.1016/j.mehy.2018.06.029 }} * {{medline-title |title=Proteomic changes during adult stage in pre-optic, hypothalamus, hippocampus and pituitary regions of female rat brain following neonatal exposure to estradiol-17β. |date=15.09.2018 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/29777688 |full-text-url=https://sci-hub.do/10.1016/j.ygcen.2018.05.005 }} * {{medline-title |title=RECQ helicase disease and related progeroid syndromes: RECQ2018 meeting. |date=07.2018 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/29752965 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6217841 }} * {{medline-title |title=Upregulation of the aging related [[LMNA]] splice variant progerin in dilated cardiomyopathy. |date=2018 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/29702688 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5922532 }} * {{medline-title |title=Increasing autophagy and blocking Nrf2 suppress laminopathy-induced age-dependent cardiac dysfunction and shortened lifespan. |date=06.2018 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/29575479 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5946079 }} * {{medline-title |title=Lipodystrophic syndromes due to [[LMNA]] mutations: recent developments on biomolecular aspects, pathophysiological hypotheses and therapeutic perspectives. |date=01.01.2018 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/29578370 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5973242 }} * {{medline-title |title=Differential stem cell aging kinetics in Hutchinson-Gilford progeria syndrome and Werner syndrome. |date=04.2018 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/29476423 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5876188 }} * {{medline-title |title=Mandibuloacral dysplasia: A premature ageing disease with aspects of physiological ageing. |date=03.2018 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/29208544 |full-text-url=https://sci-hub.do/10.1016/j.arr.2017.12.001 }} * {{medline-title |title=Hutchinson-Gilford Progeria Syndrome: A Premature Aging Disease. |date=05.2018 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/28660486 |full-text-url=https://sci-hub.do/10.1007/s12035-017-0610-7 }} * {{medline-title |title=Progerin-Induced Replication Stress Facilitates Premature Senescence in Hutchinson-Gilford Progeria Syndrome. |date=15.07.2017 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/28483909 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5492170 }} * {{medline-title |title=Physiological and Pathological Aging Affects Chromatin Dynamics, Structure and Function at the Nuclear Edge. |date=2016 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/27602048 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4993774 }} * {{medline-title |title=Clinical Trial of the Protein Farnesylation Inhibitors Lonafarnib, Pravastatin, and Zoledronic Acid in Children With Hutchinson-Gilford Progeria Syndrome. |date=12.07.2016 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/27400896 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4943677 }} * {{medline-title |title=Hutchinson-Gilford Progeria Syndrome: A premature aging disease caused by [[LMNA]] gene mutations. |date=01.2017 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/27374873 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5195863 }} * {{medline-title |title=Progeria and the early aging in children: a case report. |date=17.02.2016 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/27267192 }} * {{medline-title |title=A Heterozygous [[ZMPSTE24]] Mutation Associated with Severe Metabolic Syndrome, Ectopic Fat Accumulation, and Dilated Cardiomyopathy. |date=25.04.2016 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/27120622 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4931670 }} * {{medline-title |title=Understanding Vascular Diseases: Lessons From Premature Aging Syndromes. |date=05.2016 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/26948039 |full-text-url=https://sci-hub.do/10.1016/j.cjca.2015.12.003 }} * {{medline-title |title=Molecular insights into the premature aging disease progeria. |date=04.2016 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/26847180 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4796323 }} * {{medline-title |title=[[LMNA]] mutations resulting in lipodystrophy and HIV protease inhibitors trigger vascular smooth muscle cell senescence and calcification: Role of [[ZMPSTE24]] downregulation. |date=02.2016 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/26724531 |full-text-url=https://sci-hub.do/10.1016/j.atherosclerosis.2015.12.012 }} * {{medline-title |title=Methylene blue alleviates nuclear and mitochondrial abnormalities in progeria. |date=04.2016 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/26663466 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4783354 }} * {{medline-title |title=Pluripotent stem cells to model Hutchinson-Gilford progeria syndrome (HGPS): Current trends and future perspectives for drug discovery. |date=11.2015 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/26474742 |full-text-url=https://sci-hub.do/10.1016/j.arr.2015.10.002 }} * {{medline-title |title=All-trans retinoic acid and rapamycin normalize Hutchinson Gilford progeria fibroblast phenotype. |date=06.10.2015 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/26359359 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4745772 }} * {{medline-title |title=New look at the role of progerin in skin aging. |date=03.2015 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/26327889 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4440198 }} * {{medline-title |title=Progerin reduces LAP2α-telomere association in Hutchinson-Gilford progeria. |date=27.08.2015 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/26312502 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4565980 }} * {{medline-title |title=Overexpression of Lamin B Receptor Results in Impaired Skin Differentiation. |date=2015 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/26053873 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4459694 }} * {{medline-title |title=Sustained accumulation of prelamin A and depletion of lamin A/C both cause oxidative stress and mitochondrial dysfunction but induce different cell fates. |date=2015 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/25996284 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4615646 }} * {{medline-title |title=Age-Related Lipid Metabolic Signature in Human [[LMNA]]-Lipodystrophic Stem Cell-Derived Adipocytes. |date=07.2015 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/25961135 |full-text-url=https://sci-hub.do/10.1210/jc.2014-4528 }} * {{medline-title |title=Transgene silencing of the Hutchinson-Gilford progeria syndrome mutation results in a reversible bone phenotype, whereas resveratrol treatment does not show overall beneficial effects. |date=08.2015 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/25877214 |full-text-url=https://sci-hub.do/10.1096/fj.14-269217 }} * {{medline-title |title=Sulforaphane enhances progerin clearance in Hutchinson-Gilford progeria fibroblasts. |date=02.2015 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/25510262 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4326906 }} * {{medline-title |title=Expression of progerin in aging mouse brains reveals structural nuclear abnormalities without detectible significant alterations in gene expression, hippocampal stem cells or behavior. |date=01.03.2015 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/25343989 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4321440 }} * {{medline-title |title=Accumulation of prelamin A compromises NF-κB-regulated B-lymphopoiesis in a progeria mouse model. |date=2013 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/24764515 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3922919 }} * {{medline-title |title=Prelamin A accumulation and stress conditions induce impaired Oct-1 activity and autophagy in prematurely aged human mesenchymal stem cell. |date=04.2014 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/24753226 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4032794 }} * {{medline-title |title=Antagonistic functions of [[LMNA]] isoforms in energy expenditure and lifespan. |date=05.2014 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/24639560 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4210101 }} * {{medline-title |title=Epigenetic involvement in Hutchinson-Gilford progeria syndrome: a mini-review. |date=2014 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/24603298 |full-text-url=https://sci-hub.do/10.1159/000357206 }} * {{medline-title |title=RB and lamins in cell cycle regulation and aging. |date=2014 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/24563346 |full-text-url=https://sci-hub.do/10.1007/978-1-4899-8032-8_6 }} * {{medline-title |title=Search and insights into novel genetic alterations leading to classical and atypical Werner syndrome. |date=2014 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/24401204 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3997596 }} * {{medline-title |title=Mapping disease-related missense mutations in the immunoglobulin-like fold domain of lamin A/C reveals novel genotype-phenotype associations for laminopathies. |date=06.2014 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/24375749 |full-text-url=https://sci-hub.do/10.1002/prot.24465 }} * {{medline-title |title=Investigation of age-related changes in [[LMNA]] splicing and expression of progerin in human skeletal muscles. |date=2013 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/24294364 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3843258 }} * {{medline-title |title=Investigation of splicing changes and post-translational processing of [[LMNA]] in sporadic inclusion body myositis. |date=2013 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/24040437 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3759479 }} * {{medline-title |title=Identification of mitochondrial dysfunction in Hutchinson-Gilford progeria syndrome through use of stable isotope labeling with amino acids in cell culture. |date=08.10.2013 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/23969228 |full-text-url=https://sci-hub.do/10.1016/j.jprot.2013.08.008 }} * {{medline-title |title=The contrasting roles of lamin B1 in cellular aging and human disease. |date=07-08.2013 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/23873483 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3810336 }} * {{medline-title |title=Depleting the methyltransferase Suv39h1 improves DNA repair and extends lifespan in a progeria mouse model. |date=2013 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/23695662 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3674265 }} * {{medline-title |title=Hutchinson-Gilford progeria syndrome through the lens of transcription. |date=08.2013 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/23496208 |full-text-url=https://sci-hub.do/10.1111/acel.12070 }} * {{medline-title |title=Genetic and clinical characteristics of skeletal and cardiac muscle in patients with lamin A/C gene mutations. |date=08.2013 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/23450819 |full-text-url=https://sci-hub.do/10.1002/mus.23827 }} * {{medline-title |title=Modeling of lamin A/C mutation premature cardiac aging using patient‐specific induced pluripotent stem cells. |date=11.2012 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/23362510 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3560431 }} * {{medline-title |title=Aberrant DNA methylation profiles in the premature aging disorders Hutchinson-Gilford Progeria and Werner syndrome. |date=01.2013 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/23257959 |full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3549877 }} * {{medline-title |title=Resveratrol rescues [[SIRT1]]-dependent adult stem cell decline and alleviates progeroid features in laminopathy-based progeria. |date=05.12.2012 |pubmed-url=https://pubmed.ncbi.nlm.nih.gov/23217256 |full-text-url=https://sci-hub.do/10.1016/j.cmet.2012.11.007 }}
Описание изменений:
Пожалуйста, учтите, что любой ваш вклад в проект «hpluswiki» может быть отредактирован или удалён другими участниками. Если вы не хотите, чтобы кто-либо изменял ваши тексты, не помещайте их сюда.
Вы также подтверждаете, что являетесь автором вносимых дополнений, или скопировали их из источника, допускающего свободное распространение и изменение своего содержимого (см.
Hpluswiki:Авторские права
).
НЕ РАЗМЕЩАЙТЕ БЕЗ РАЗРЕШЕНИЯ ОХРАНЯЕМЫЕ АВТОРСКИМ ПРАВОМ МАТЕРИАЛЫ!
Отменить
Справка по редактированию
(в новом окне)