FADD

Материал из hpluswiki
Перейти к навигации Перейти к поиску

FAS-associated death domain protein (FAS-associating death domain-containing protein) (Growth-inhibiting gene 3 protein) (Mediator of receptor induced toxicity) (Protein FADD) [MORT1] [GIG3]

Publications[править]

Voluntary exercise training attenuated the middle-aged maturity-induced cardiac apoptosis.


[[[FAS]]- and TNF-dependent ways participation in apoptosis mechanisms in hypotalumus in physiological and pathological aging.]


Anti-Apoptotic Effects of Diosgenin in D-Galactose-Induced Aging Brain.


Improved age-related deficits in cognitive performance and affective-like behavior following acute, but not repeated, 8-OH-DPAT treatments in rats: regulation of hippocampal FADD.


Decreased cortical FADD protein is associated with clinical dementia and cognitive decline in an elderly community sample.


Chronic MDMA induces neurochemical changes in the hippocampus of adolescent and young adult rats: Down-regulation of apoptotic markers.


The Heart Protection Effect of Alcalase Potato Protein Hydrolysate Is through IGF1R-PI3K-Akt Compensatory Reactivation in Aging Rats on High Fat Diets.


TNF/TNFR₁ pathway and endoplasmic reticulum stress are involved in ofloxacin-induced apoptosis of juvenile canine chondrocytes.


Epigallocatechin-3-gallate improves plantaris muscle recovery after disuse in aged rats.


A critical role of Fas-associated protein with death domain phosphorylation in intracellular reactive oxygen species homeostasis and aging.