BMI1

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Polycomb complex protein BMI-1 (Polycomb group RING finger protein 4) (RING finger protein 51) [PCGF4] [RNF51]

Publications[править]

Senescence Induced by BMI1 Inhibition Is a Therapeutic Vulnerability in H3K27M-Mutant DIPG.


microRNA-31 inhibition partially ameliorates the deficiency of bone marrow stromal cells from cleidocranial dysplasia.


Timosaponin A-III inhibits oncogenic phenotype via regulation of PcG protein BMI1 in breast cancer cells.


Suppressing P16 and P14 pathways overcomes apoptosis in individualized human embryonic stem cells.


BMI1 inhibits senescence and enhances the immunomodulatory properties of human mesenchymal stem cells via the direct suppression of MKP-1/DUSP1.


Adipose-derived stem cells from lean and obese humans show depot specific differences in their stem cell markers, exosome contents and senescence: role of protein kinase C delta (PKCδ) in adipose stem cell niche.


A miR-200c/141-BMI1 autoregulatory loop regulates oncogenic activity of BMI1 in cancer cells.


MicroRNA-31 is a transcriptional target of histone deacetylase inhibitors and a regulator of cellular senescence.


PLK1 inhibition down-regulates polycomb group protein BMI1 via modulation of the miR-200c/141 cluster.


microRNA-141 regulates BMI1 expression and induces senescence in human diploid fibroblasts.