Редактирование: Публикации о генах и старении (titles) (раздел)

==TET2==

{{medline-entry
|title=Non-coding and Loss-of-Function Coding Variants in [[TET2]] are Associated with Multiple Neurodegenerative Diseases.
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/32330418

|mesh-terms=* Aged
* Aged, 80 and over
* Alzheimer Disease
* Animals
* Cognition
* DNA-Binding Proteins
* Female
* Frontotemporal Dementia
* Humans
* Loss of Function Mutation
* Male
* Mice
* Neurodegenerative Diseases
* Proto-Oncogene Proteins
|keywords=* AD
* ALS
* Alzheimer
* FTD
* TET2
* aging
* amyotrophic lateral sclerosis
* frontotemporal dementia
* genome sequencing
* non-coding
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7212268
}}
{{medline-entry
|title=60 Years of clonal hematopoiesis research: From X-chromosome inactivation studies to the identification of driver mutations.
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/32001340

|mesh-terms=* Adult
* Aging
* Biomedical Research
* Chromosomes, Human, X
* DNA-Binding Proteins
* Female
* Hematopoiesis
* Hematopoietic Stem Cells
* History, 20th Century
* History, 21st Century
* Humans
* Male
* Mutation
* Proto-Oncogene Proteins
* Receptors, Androgen
* Repressor Proteins
* X Chromosome Inactivation

|full-text-url=https://sci-hub.do/10.1016/j.exphem.2020.01.008
}}
{{medline-entry
|title=DNA methylation instability by BRAF-mediated TET silencing and lifestyle-exposure divides colon cancer pathways.
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/31842975

|mesh-terms=* Animals
* Caco-2 Cells
* Cell Line, Tumor
* Colonic Neoplasms
* DNA Methylation
* DNA-Binding Proteins
* Down-Regulation
* Epigenesis, Genetic
* Female
* Gene Regulatory Networks
* HT29 Cells
* Humans
* Male
* Mice
* Mixed Function Oxygenases
* Mutation
* Neoplasms, Experimental
* Proto-Oncogene Proteins
* Proto-Oncogene Proteins B-raf
|keywords=* Aging
* BRAF V600E
* CIMP
* Colon cancer
* DNA methylation
* TET
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6916434
}}
{{medline-entry
|title=Clonal haematopoiesis: connecting ageing and inflammation in cardiovascular disease.
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/31406340

|mesh-terms=* Adult
* Age Factors
* Aged
* Aged, 80 and over
* Aging
* Animals
* Cardiovascular Diseases
* DNA (Cytosine-5-)-Methyltransferases
* DNA-Binding Proteins
* Genetic Predisposition to Disease
* Hematopoiesis
* Hematopoietic Stem Cells
* Humans
* Inflammation
* Middle Aged
* Mutation
* Phenotype
* Proto-Oncogene Proteins
* Repressor Proteins
* Risk Assessment
* Risk Factors

|full-text-url=https://sci-hub.do/10.1038/s41569-019-0247-5
}}