Редактирование: Aging genes 20-29 (раздел)

==PML==

* {{medline-title
|title=Progressive multifocal leukoencephalopathy in dimethyl fumarate-treated multiple sclerosis patients.
|date=18.08.2020
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/32808554
|full-text-url=https://sci-hub.do/10.1177/1352458520949158
}}
* {{medline-title
|title=[[PML]]2-mediated thread-like nuclear bodies mark late senescence in Hutchinson-Gilford progeria syndrome.
|date=06.2020
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/32351002
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7294779
}}
* {{medline-title
|title=Trophoblast type-specific expression of senescence markers in the human placenta.
|date=15.09.2019
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/31327484
|full-text-url=https://sci-hub.do/10.1016/j.placenta.2019.06.377
}}
* {{medline-title
|title=[[PML]] nuclear bodies are recruited to persistent DNA damage lesions in an [[RNF168]]-53BP1 dependent manner and contribute to DNA repair.
|date=06.2019
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/31009828
|full-text-url=https://sci-hub.do/10.1016/j.dnarep.2019.04.001
}}
* {{medline-title
|title=Nuclear Nox4 interaction with prelamin A is associated with nuclear redox control of stem cell aging.
|date=24.10.2018
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/30362963
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6224265
}}
* {{medline-title
|title=Aging and lymphocyte changes by immunomodulatory therapies impact [[PML]] risk in multiple sclerosis patients.
|date=07.2018
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/29774781
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6013383
}}
* {{medline-title
|title=[[SOCS1]] regulates senescence and ferroptosis by modulating the expression of p53 target genes.
|date=28.10.2017
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/29081404
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5680560
}}
* {{medline-title
|title=Age as a risk factor for early onset of natalizumab-related progressive multifocal leukoencephalopathy.
|date=10.2017
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/28791614
|full-text-url=https://sci-hub.do/10.1007/s13365-017-0561-9
}}
* {{medline-title
|title=The Role of Sumoylation in Senescence.
|date=2017
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/28197915
|full-text-url=https://sci-hub.do/10.1007/978-3-319-50044-7_13
}}
* {{medline-title
|title=Epigenetic Aging Signatures Are Coherently Modified in Cancer.
|date=06.2015
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/26110659
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4482318
}}
* {{medline-title
|title=Downregulation of PEA-15 reverses G1 arrest, and nuclear and chromatin changes of senescence phenotype via pErk1/2 translocation to nuclei.
|date=06.2015
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/25725291
|full-text-url=https://sci-hub.do/10.1016/j.cellsig.2015.02.014
}}
* {{medline-title
|title=[[PML]] nuclear bodies: assembly and oxidative stress-sensitive sumoylation.
|date=2014
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/25482067
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4615786
}}
* {{medline-title
|title=Wnt5A promotes an adaptive, senescent-like stress response, while continuing to drive invasion in melanoma cells.
|date=03.2015
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/25407936
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4333017
}}
* {{medline-title
|title=[[PML]] nuclear bodies: regulation, function and therapeutic perspectives.
|date=11.2014
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/25138686
|full-text-url=https://sci-hub.do/10.1002/path.4426
}}
* {{medline-title
|title=Rapamycin prevents strong phosphorylation of p53 on serine 46 and attenuates activation of the p53 pathway in A549 lung cancer cells exposed to actinomycin D.
|date=07.2014
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/24915467
|full-text-url=https://sci-hub.do/10.1016/j.mad.2014.06.002
}}
* {{medline-title
|title=[[PML]]-nuclear bodies decrease with age and their stress response is impaired in aged individuals.
|date=02.04.2014
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/24694011
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3992156
}}
* {{medline-title
|title=The retinoblastoma protein and [[PML]] collaborate to organize heterochromatin and silence E2F-responsive genes during senescence.
|date=2014
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/24351540
|full-text-url=https://sci-hub.do/10.4161/cc.27527
}}
* {{medline-title
|title=Dynamics of histone H3.3 deposition in proliferating and senescent cells reveals a [[DAXX]]-dependent targeting to [[PML]]-NBs important for pericentromeric heterochromatin organization.
|date=2014
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/24200965
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3906242
}}
* {{medline-title
|title=Material properties of aged human mitral valve leaflets.
|date=08.2014
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/24039052
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4033712
}}
* {{medline-title
|title=[[PML]], SUMOylation, and Senescence.
|date=2013
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/23847762
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3701148
}}
* {{medline-title
|title=[[DAXX]] silencing suppresses mouse ovarian surface epithelial cell growth by inducing senescence and DNA damage.
|date=10.09.2013
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/23542781
|full-text-url=https://sci-hub.do/10.1016/j.gene.2013.03.103
}}
* {{medline-title
|title=On [[BLM]] helicase in recombination-mediated telomere maintenance.
|date=04.2013
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/23268311
|full-text-url=https://sci-hub.do/10.1007/s11033-012-2379-0
}}