Редактирование: Aging genes 5-9 (раздел)

==XIAP==

* {{medline-title
|title=Directed elimination of senescent cells attenuates development of osteoarthritis by inhibition of c-IAP and [[XIAP]].
|date=01.10.2019
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/31251987
|full-text-url=https://sci-hub.do/10.1016/j.bbadis.2019.05.017
}}
* {{medline-title
|title=Modulated electro-hyperthermia induced p53 driven apoptosis and cell cycle arrest additively support doxorubicin chemotherapy of colorectal cancer in vitro.
|date=08.2019
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/31183995
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6675742
}}
* {{medline-title
|title=Protective effect of xanthohumol against age-related brain damage.
|date=11.2017
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/28950154
|full-text-url=https://sci-hub.do/10.1016/j.jnutbio.2017.07.011
}}
* {{medline-title
|title=Molecular modeling in the age of clinical genomics, the enterprise of the next generation.
|date=03.2017
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/28204942
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5529140
}}
* {{medline-title
|title=Sperm-associated antigen 9 ([[SPAG9]]) promotes the survival and tumor growth of triple-negative breast cancer cells.
|date=10.2016
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/27449044
|full-text-url=https://sci-hub.do/10.1007/s13277-016-5240-6
}}
* {{medline-title
|title=[[XIAP]]-associating factor 1, a transcriptional target of [[BRD7]], contributes to endothelial cell senescence.
|date=02.02.2016
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/26802028
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4868675
}}
* {{medline-title
|title=Targeting protein neddylation with an [[NEDD8]]-activating enzyme inhibitor MLN4924 induced apoptosis or senescence in human lymphoma cells.
|date=2015
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/25782162
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4623239
}}
* {{medline-title
|title=Increase in retinal ganglion cells' susceptibility to elevated intraocular pressure and impairment of their endogenous neuroprotective mechanism by age.
|date=2013
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/24146536
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3783363
}}
* {{medline-title
|title=The member of high temperature requirement family HtrA2 participates in neuronal apoptosis after intracerebral hemorrhage in adult rats.
|date=08.2013
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/23413020
|full-text-url=https://sci-hub.do/10.1007/s10735-013-9489-4
}}