Редактирование: Публикации о генах и старении (titles) (раздел)

==ERCC1==

{{medline-entry
|title=Chronic Sildenafil Treatment Improves Vasomotor Function in a Mouse Model of Accelerated Aging.
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/32630010


|keywords=* aging
* cGMP
* guanylate cyclase
* hypertension
* nitric oxide
* phosphodiesterase
* sildenafil
* vascular dysfunction
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7369923
}}
{{medline-entry
|title=Local endothelial DNA repair deficiency causes aging-resembling endothelial-specific dysfunction.
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/32202295

|mesh-terms=* Age Factors
* Aging
* Animals
* Capillary Permeability
* Cellular Senescence
* Cyclin-Dependent Kinase Inhibitor p21
* DNA Damage
* DNA Repair
* DNA-Binding Proteins
* Endonucleases
* Endothelial Cells
* Endothelium, Vascular
* Mice, Inbred C57BL
* Mice, Knockout
* Nitric Oxide
* Nitric Oxide Synthase Type III
* Superoxides
* Vascular Stiffness
* Vasodilation
|keywords=* DNA damage
* aging
* endothelial dysfunction
* endothelium-dependent dilation
* nitric oxide
|full-text-url=https://sci-hub.do/10.1042/CS20190124
}}
{{medline-entry
|title=Tissue specificity of senescent cell accumulation during physiologic and accelerated aging of mice.
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/31981461


|keywords=* DNA repair
* ERCC1-XPF
* aging
* cellular senescence
* endogenous DNA damage
* progeria
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7059165
}}
{{medline-entry
|title=Deficiency in the DNA repair protein [[ERCC1]] triggers a link between senescence and apoptosis in human fibroblasts and mouse skin.
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/31737985


|keywords=* DNA damage repair
* aging
* cell death
* senescence-associated secretory phenotype
* tumor necrosis factor α
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7059167
}}