Редактирование: Aging genes 5-9 (раздел)

==CLN3==

* {{medline-title
|title=[[SIRT6]] histone deacetylase functions as a potential oncogene in human melanoma.
|date=09.2017
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/29234488
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5724804
}}
* {{medline-title
|title=Age-dependent alterations in neuronal activity in the hippocampus and visual cortex in a mouse model of Juvenile Neuronal Ceroid Lipofuscinosis ([[CLN3]]).
|date=04.2017
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/28042098
|full-text-url=https://sci-hub.do/10.1016/j.nbd.2016.12.022
}}
* {{medline-title
|title=Photoreceptor phagosome processing defects and disturbed autophagy in retinal pigment epithelium of Cln3Δex1-6 mice modelling juvenile neuronal ceroid lipofuscinosis (Batten disease).
|date=15.12.2015
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/26450516
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4654058
}}
* {{medline-title
|title=Finding the most appropriate mouse model of juvenile [[CLN3]] (Batten) disease for therapeutic studies: the importance of genetic background and gender.
|date=04.2015
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/26035843
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4381334
}}
* {{medline-title
|title=Xbp1 directs global repression of budding yeast transcription during the transition to quiescence and is important for the longevity and reversibility of the quiescent state.
|date=10.2013
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/24204289
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3814307
}}