Редактирование: Aging genes 10-19 (раздел)

==CCL11==

* {{medline-title
|title=CCL-11 or Eotaxin-1: An Immune Marker for Ageing and Accelerated Ageing in Neuro-Psychiatric Disorders.
|date=02.09.2020
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/32887304
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7558796
}}
* {{medline-title
|title=Circulating factors in young blood as potential therapeutic agents for age-related neurodegenerative and neurovascular diseases.
|date=11.2019
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/31400495
|full-text-url=https://sci-hub.do/10.1016/j.brainresbull.2019.08.004
}}
* {{medline-title
|title=Young bone marrow transplantation preserves learning and memory in old mice.
|date=2019
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/30820468
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6382867
}}
* {{medline-title
|title=[The role of «Youth and aging proteins» in essential hypertension pathogenesis.]
|date=2018
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/30584875

}}
* {{medline-title
|title=Association between plasma [[CCL11]] (eotaxin-1) and cognitive status in older adults: Differences between rural and urban dwellers.
|date=11.2018
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/30308289
|full-text-url=https://sci-hub.do/10.1016/j.exger.2018.10.004
}}
* {{medline-title
|title=Inflammaging impairs peripheral nerve maintenance and regeneration.
|date=12.2018
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/30168637
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6260910
}}
* {{medline-title
|title=Stem Cell-Induced Pulp Regeneration Can Be Enhanced by Administration of [[CCL11]]-Neutralizing Antibody in the Ectopic Tooth Transplantation Model in the Aged Mice.
|date=02.2019
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/29962294
|full-text-url=https://sci-hub.do/10.1089/rej.2018.2064
}}
* {{medline-title
|title=Inflammatory chemokine eotaxin-1 is correlated with age in heroin dependent patients under methadone maintenance therapy.
|date=01.02.2018
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/29222992
|full-text-url=https://sci-hub.do/10.1016/j.drugalcdep.2017.10.014
}}
* {{medline-title
|title=["Protein of senility" [[CCL11]], "protein of juvenility" [[GDF11]] and their role in age-related pathology].
|date=2016
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/28556640

}}
* {{medline-title
|title=Telomere Length and [[CCL11]] Levels are Associated With Gray Matter Volume and Episodic Memory Performance in Schizophrenia: Evidence of Pathological Accelerated Aging.
|date=13.01.2018
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/28338779
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5767949
}}
* {{medline-title
|title=An emerging role for eotaxins in neurodegenerative disease.
|date=04.2018
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/27664933
|full-text-url=https://sci-hub.do/10.1016/j.clim.2016.09.010
}}
* {{medline-title
|title=Blood-Borne Revitalization of the Aged Brain.
|date=10.2015
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/26237737
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4867550
}}
* {{medline-title
|title=[Peptides and [[CCL11]] and [[HMGB1]] as molecular markers of aging: literature review and own data].
|date=2014
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/25826983

}}
* {{medline-title
|title=Rejuvenation: it's in our blood.
|date=01.07.2014
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/24988454
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4126586
}}
* {{medline-title
|title=Effect of high-intensity exercise on aged mouse brain mitochondria, neurogenesis, and inflammation.
|date=11.2014
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/25002036
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4171347
}}
* {{medline-title
|title=Cannabis use is associated with increased [[CCL11]] plasma levels in young healthy volunteers.
|date=01.10.2013
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/23820464
|full-text-url=https://sci-hub.do/10.1016/j.pnpbp.2013.06.011
}}
* {{medline-title
|title=CNS-specific immunity at the choroid plexus shifts toward destructive Th2 inflammation in brain aging.
|date=05.02.2013
|pubmed-url=https://pubmed.ncbi.nlm.nih.gov/23335631
|full-text-url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3568380
}}